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This unique structure allows GHK-Cu to function both as a copper delivery system and as a signaling molecule capable of modulating cellular activities
The half-life is too long
Our previous study has shown that MAPK and Akt signaling markedly upregulated in acute myocardial infarction in porcine that was suppressed by tacrolimus treatment.[37] Interestingly, in the in vivo studies, we also found that the PI3K/p-AKT/m-TOR signaling pathway was markedly upregulated in F344 HS animals than in SC counterparts, suggesting an intrinsic response to ischemic stimulation
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